영어 모의고사 (자동 편성)

Political philosophers have long debated how much authority, if any, a government may ①legitimate claim over free individuals. Robert Nozick imagined a hypothetical starting point in which people live without any state at all, each relying on private arrangements to protect their own person and property. He argued that a minimal state, limited strictly to enforcing contracts and protecting citizens from violence, theft, and fraud, could ②arise gradually through a series of voluntary exchanges, without anyone's rights ever being violated along the way. Critics on one side, the anarchists, insist that even this minimal government inevitably ③violates the rights of those who never consented to join it. Critics on the other side argue that a state limited to bare protection cannot ④address deeper inequalities that arise naturally in any free market. Nozick's thought experiment therefore sits at the center of an unresolved argument about where legitimate political authority actually ⑤begins.

Imagine rewriting a bacterium's code the way a programmer ①edits a line of software, and much of what synthetic biology promises begins to make sense. Researchers in this field assemble standardized genetic parts into circuits ②that can be inserted into a cell, instructing it to perform a task that no natural organism would carry out. Because these circuits are built from interchangeable parts rather than designed from scratch each time, a circuit tested in one lab can often ③reusing, with minor adjustments, in an entirely different application. Critics note that biological systems rarely behave as predictably as electronic circuits, since a gene ④inserted into a new cell can interact with existing pathways in unexpected ways, occasionally producing results no engineer intended. Even so, as the catalog of standardized parts keeps expanding, engineers are gaining confidence ⑤to design increasingly complex systems, treating the cell as a chassis onto which new functions can be loaded.

When individuals face uncertainty about how to behave, they frequently look to the actions of others for guidance. This reliance on collective behavior can be remarkably ①efficient, since it allows individuals to benefit from the accumulated judgment of a crowd without having to gather and evaluate every relevant fact themselves. However, social proof can also lead entire groups astray when the initial signal that triggers imitation is itself ②unreliable or coincidental. A restaurant that happens to attract a long line early in the evening may continue attracting customers throughout the night simply because passersby interpret the queue as evidence of ③inferior quality. Financial markets exhibit an analogous pattern: investors who observe others buying a particular asset often join the buying themselves, reasoning that so many participants cannot all be mistaken, a dynamic that can ④inflate prices well beyond any reasonable estimate of underlying value. Because conformity can substitute for independent judgment so ⑤seamlessly, individuals rarely notice when their own decisions have become mere imitations rather than genuinely deliberated.

A liver no longer has to sit inside a human body to be studied closely. By coaxing stem cells to organize themselves within a nutrient-rich gel, scientists can now grow miniature three-dimensional structures called organoids, which reproduce much of the architecture and function of a real organ. Unlike flat cultures grown on a plastic dish, organoids let cells interact in three dimensions much as they would inside the body, yielding tissue that behaves in strikingly realistic ways. This fidelity has made organoids invaluable for testing how a drug affects human tissue before any patient is exposed, _______________. Some laboratories have even grown organoids from a single patient's own cells, raising the possibility of trialing a treatment on a miniature copy of that patient's organ before committing to an actual course of therapy.

When an oil tanker ruptures and crude spreads across open water, cleanup crews increasingly reach not only for skimmers and floating booms but also for something far smaller: bacteria that eat oil. Certain naturally occurring microbes possess enzymes capable of breaking hydrocarbon molecules apart, using the carbon as an energy source and gradually converting a toxic slick into carbon dioxide and harmless byproducts. Bioremediation, as this approach is called, works far more slowly than mechanical cleanup, often taking months rather than days, but it can reach contamination trapped in soil or sediment that skimmers and booms can never reach. Scientists have learned to accelerate the process by adding nutrients that help native microbial populations multiply faster, or in some cases by introducing engineered strains selected specifically for their appetite for a particular pollutant. _______________.

The role of emotion in decision-making carries important implications for how financial advice should be delivered. (A) An advisor who presents only statistical projections and probability distributions, however accurate, may fail to help a client translate that information into a decision that feels manageable rather than overwhelming. (B) Effective financial communication, therefore, often pairs quantitative analysis with reassurance calibrated to the client's emotional state, particularly during periods of market turbulence. (C) Clients facing significant financial decisions are rarely persuaded by numbers alone, since anxiety about an uncertain outcome can override even a clear rational case for a particular course of action.

Bacteria have been cutting up invading viral DNA for billions of years, long before any scientist thought to borrow the trick for use in a laboratory. (A) Once Cas9 arrives at the target site, it slices both strands of the DNA helix, and the cell's own repair machinery rushes in to mend the cut, often introducing a small change in the process. (B) By supplying the cell with a template during this repair step, scientists can direct that machinery to insert a specific new sequence, and what once required years of painstaking work to alter a single gene can now be accomplished within days. (C) The CRISPR-Cas9 system, as it is now known, relies on a short guide RNA that matches a specific DNA sequence, leading a scissor-like enzyme called Cas9 directly to that exact location in the genome.

For decades, most vaccines worked by injecting a weakened or inactivated version of a pathogen, coaxing the immune system into recognizing an enemy it had never truly fought. (A) mRNA vaccines take a fundamentally different route, delivering only a short genetic instruction wrapped in a protective lipid capsule, one that tells the body's own cells how to manufacture a single harmless piece of a virus's outer surface protein. (B) Once those cells display the fragment, the immune system learns to recognize it as foreign and builds a defense, without ever having encountered the actual pathogen. (C) Because the vaccine contains no live virus and cannot alter a cell's own DNA, developers can redesign the genetic instructions within weeks whenever a pathogen mutates, a speed that has convinced many researchers the same platform could eventually fight diseases far beyond infectious illness.

Two patients diagnosed with the same disease and prescribed the same medication can respond in strikingly different ways, one recovering quickly while the other experiences little benefit or even harmful side effects. (A) This approach has proven especially valuable in cancer treatment, where tumors from two patients with the same diagnosis may carry entirely different genetic mutations that respond to entirely different drugs, though widespread adoption still faces real obstacles, since genetic sequencing remains expensive in many regions. (B) By sequencing a patient's DNA before treatment begins, doctors can increasingly predict which medication is likely to work best and at what dose, replacing a strategy once described as one-size-fits-all with something closer to a tailored fit. (C) Precision medicine starts from the observation that this variation often traces back to differences buried in each patient's genome, differences that determine how efficiently a body absorbs, breaks down, or responds to a given drug.

In other instances, the danger came not from the immune system at all but from where the new gene happened to land. For patients born with a single faulty gene, the disease has traditionally been treated indirectly, managing symptoms for a lifetime rather than addressing the genetic error itself. ( ① ) Gene therapy attempts something more ambitious: delivering a corrected copy of the gene directly into a patient's cells, often using a disabled virus stripped of its ability to cause illness as a delivery vehicle. ( ② ) Once inside the nucleus, the corrected gene can begin producing the protein the patient's body had always lacked, in some cases reversing symptoms that medication alone had never been able to touch. ( ③ ) Early trials were haunted by serious setbacks, including cases in which the delivery virus triggered a dangerous immune reaction. ( ④ ) The virus, in such cases, inserted the new gene in a spot that disrupted other essential genes. ( ⑤ ) Refined delivery methods have since made the treatment considerably safer, and regulators in several countries have now approved gene therapies for a small but growing number of rare inherited disorders.