영어 모의고사 (자동 편성)

Imagine rewriting a bacterium's code the way a programmer ①edits a line of software, and much of what synthetic biology promises begins to make sense. Researchers in this field assemble standardized genetic parts into circuits ②that can be inserted into a cell, instructing it to perform a task that no natural organism would carry out. Because these circuits are built from interchangeable parts rather than designed from scratch each time, a circuit tested in one lab can often ③reusing, with minor adjustments, in an entirely different application. Critics note that biological systems rarely behave as predictably as electronic circuits, since a gene ④inserted into a new cell can interact with existing pathways in unexpected ways, occasionally producing results no engineer intended. Even so, as the catalog of standardized parts keeps expanding, engineers are gaining confidence ⑤to design increasingly complex systems, treating the cell as a chassis onto which new functions can be loaded.

Before a large-scale construction project such as a highway or dam receives government approval, planners in many countries are required to conduct an environmental impact assessment, a formal process designed to ①predict and evaluate the ecological consequences of a proposed development. This process begins with a baseline survey, in which specialists document ②existing conditions, including wildlife, water quality, and air composition, so that future changes can be measured against a reference point. Analysts then model how the project might alter these conditions, drawing on data from ③comparable developments. For instance, a proposed highway that would bisect a forest might be evaluated for its potential to fragment habitats, disrupting migration routes on which certain species depend. If the assessment identifies ④acceptable risks, planners may be required to redesign the project or adopt mitigation measures such as wildlife corridors. Critics contend, however, that assessments are sometimes treated as a bureaucratic formality rather than a ⑤genuine constraint, since developers who commission the studies retain strong influence over how the findings are interpreted.

A biosensor works by pairing a biological component, such as an enzyme or antibody, with an electronic device that converts a biological reaction into a signal a computer can ①read. When the target molecule, whether glucose in blood or a trace of a pathogen, binds to the biological component, it triggers a ②massive measurable change that the device translates into a readable number. This design has already transformed diabetes management, letting patients monitor blood sugar ③continuously through a small sensor worn under the skin rather than pricking a finger several times a day. Engineers are now racing to extend the same principle to detect subtler targets, from cancer cells circulating in the bloodstream to trace ④contaminants in drinking water. The central challenge is not detecting the target but doing so reliably outside a controlled laboratory, since a sensor that performs well on a clean lab bench can behave ⑤erratically once exposed to a real body or a real river.

For decades, most vaccines worked by injecting a ①weakened or inactivated version of a pathogen, coaxing the immune system into recognizing an enemy it had never truly fought. mRNA vaccines take a fundamentally ②different route, delivering only a short genetic instruction wrapped in a protective lipid capsule, one that tells the body's own cells how to manufacture a single harmless piece of a virus's outer surface protein. Once those cells display the fragment, the immune system learns to recognize it as ③foreign and builds a defense, without ever having encountered the actual pathogen. Because the vaccine contains no live virus and cannot alter a cell's own DNA, developers can ④redesign the genetic instructions within weeks whenever a pathogen mutates, rather than starting the years-long process required for traditional vaccines. This speed has convinced many researchers that the same platform could eventually be adapted to fight diseases far beyond infectious illness, ⑤excluding certain cancers.

As solar and wind power expand rapidly across national grids, engineers face a challenge that receives far less public attention than the construction of the generating facilities themselves. Unlike conventional power plants, which can adjust their output to match demand at almost any moment, solar panels and wind turbines generate electricity only when sunlight or wind is available, producing supply that fluctuates constantly. This intermittency means that grid operators must balance supply and demand even when renewable output suddenly drops, such as when clouds cover a solar farm or wind speeds fall. One proposed solution involves large-scale battery storage systems, which absorb surplus electricity generated during periods of high output and release it later when generation declines. Another approach relies on expanding transmission networks across wider regions, so that electricity generated in a sunny area can be redirected to places experiencing a shortfall. Grid operators _______________. Without such investment, integrating renewable energy could paradoxically undermine the very reliability that modern electricity grids are expected to provide.

Political philosophers have long debated how much authority, if any, a government may legitimately claim over free individuals. Robert Nozick imagined a hypothetical starting point in which people live without any state at all, each relying on private arrangements to protect their own person and property. He argued that a minimal state, limited strictly to enforcing contracts and protecting citizens from violence, theft, and fraud, could arise gradually through a series of voluntary exchanges, without anyone's rights ever being violated along the way. Critics on one side, the anarchists, insist that even this minimal government inevitably violates the rights of those who never consented to join it. Critics on the other side argue that a state limited to bare protection cannot address deeper inequalities that arise naturally in any free market. Nozick's thought experiment therefore _______________.

John Rawls proposed an unusual thought experiment to determine what principles of justice a society ought to adopt. (A) Imagine a group of rational people gathered to design the basic rules of their future society, but placed behind what he called a veil of ignorance, unaware of their own race, gender, wealth, or social position. (B) Since no one can predict whether they will be born into privilege or poverty, each person has a strong incentive to choose rules that protect the worst-off members of society. (C) Rawls argued that rational people placed in this position would favor arrangements that guarantee basic liberties for everyone and permit unequal outcomes only when they benefit the least advantaged.

When a resource is shared among many users, none of whom bears the full cost of its depletion, each individual is tempted to extract as much value as possible before others do the same. (A) This dynamic, famously labeled the tragedy of the commons, describes how shared pastures and fisheries can be degraded even when every participant privately understands the danger of overuse. (B) A single herder who adds one more animal to a communal pasture gains the entire benefit of that addition, whereas the resulting damage to the grass is shared among other herders who depend on the land. Because self-interest offers no incentive to restrain consumption unilaterally, the resource tends to be exhausted long before any user intends such an outcome. (C) For decades, economists assumed that only two remedies existed: strict regulation or the conversion of shared resources into private property. Elinor Ostrom challenged this assumption, however, by documenting communities that had sustainably managed fisheries and forests for generations through locally devised rules and mutual monitoring rather than external control.

The lining, weakened in this way, no longer functions as the tight barrier it is meant to be. The human gut hosts trillions of microorganisms whose combined genetic material outnumbers that of the human genome itself many times over. ( ① ) Once dismissed as passive hitchhikers, these bacteria are now understood to actively shape digestion, regulate immune responses, and even influence the production of chemicals that affect mood and behavior. ( ② ) A microbiome disrupted by antibiotics, poor diet, or chronic stress can leave the gut lining more permeable than it should be. ( ③ ) This allows substances to leak into the bloodstream that would normally stay contained. ( ④ ) Researchers studying this connection have found that transplanting gut bacteria from a healthy donor into a patient can, in some cases, relieve symptoms of certain digestive disorders that conventional medication had failed to treat. ( ⑤ ) Still, scientists caution that the microbiome varies enormously between individuals, which means a treatment that restores balance in one person's gut may do little for someone else's.

Turning this tide has proven far harder than creating it, and not merely for scientific reasons. Every time an antibiotic kills off a population of bacteria, a handful of cells carrying a random mutation that resists the drug are left behind to multiply without competition. ( ① ) Repeated over decades, across hospitals, farms, and households worldwide, this simple process of natural selection has produced strains of bacteria that survive treatments once considered reliably effective. ( ② ) The pharmaceutical industry, meanwhile, has slowed the pace at which new antibiotics reach the market, since a drug meant to be taken for only a week or two rarely earns back the enormous cost of developing it. ( ③ ) Researchers are therefore searching in unconventional places, screening soil microbes and ocean sediment for molecules that kill bacteria through new mechanisms. ( ④ ) Even genetic databases are now mined for such molecules, ones resistant strains have not yet learned to evade. ( ⑤ ) Some scientists have also revived an older approach, using viruses that infect only bacteria to hunt down resistant strains with a precision no broad-spectrum drug could ever match.